{"id":29258,"date":"2025-07-16T16:59:46","date_gmt":"2025-07-16T14:59:46","guid":{"rendered":"https:\/\/www.neolifesalud.com\/blog\/sin-categorizar\/longevitat-intelligent-claus-moleculars-per-envellir-millor\/"},"modified":"2025-07-16T16:59:46","modified_gmt":"2025-07-16T14:59:46","slug":"longevitat-intelligent-claus-moleculars-per-envellir-millor","status":"publish","type":"post","link":"https:\/\/www.neolifesalud.com\/ca\/blog\/neolife-ca\/longevitat-intelligent-claus-moleculars-per-envellir-millor\/","title":{"rendered":"Longevitat intel\u00b7ligent: claus moleculars per envellir millor"},"content":{"rendered":"
\n

Es pot “modular” l’envelliment? La resposta \u00e9s: S\u00ed. I la ci\u00e8ncia ens d\u00f3na cada cop m\u00e9s eines per fer-ho de forma segura i efectiva. Durant anys es va assumir que l’envelliment era un proc\u00e9s inevitable, dictat exclusivament per la gen\u00e8tica i el pas del temps. <\/strong><\/h1>\n

Avui sabem que no \u00e9s aix\u00ed. La investigaci\u00f3 biom\u00e8dica ha identificat m\u00faltiples rutes metab\u00f2liques i moleculars que poden ser influenciades activament per alentir el deteriorament fisiol\u00f2gic, augmentar la vitalitat i preservar la funci\u00f3 cognitiva i f\u00edsica fins i tot en edats avan\u00e7ades. <\/p>\n

Dra. Y\u00e9ssica S\u00e1nchez – Equip M\u00e8dic Neolife <\/em><\/p>\n

\n

Claus moleculars que revolucionen l’enfocament cl\u00ednic de l’envelliment saludable<\/strong><\/p>\n

La longevitat<\/strong> ja no \u00e9s nom\u00e9s una q\u00fcesti\u00f3 d’anys, sin\u00f3 de qualitat de vida<\/strong>: energia sostinguda, metabolisme actiu, funci\u00f3 cognitiva preservada, baix nivell d’inflamaci\u00f3, per\u00f2 sobretot mantenir-se sense malaltia. Un dels pilars d’aquest enfocament \u00e9s el manteniment de l’equilibri cel\u00b7lular a nivell d’oxidaci\u00f3 i reducci\u00f3 per evitar la inflamaci\u00f3; hi ha proves per valorar aquest equilibri mesurant l’activitat de diferents enzims i els seus metab\u00f2lits. A Neolife ho mesurem tant a la sang com a les c\u00e8l\u00b7lules per tal d’estructurar una pauta personalitzada per aconseguir el correcte equilibri i funci\u00f3 cel\u00b7lular. <\/p>\n

\"envelliment<\/p>\n

En aquest tipus destudis destaquen antioxidants<\/strong> end\u00f2gens que tenen un paper summament important en la salut: el glutati\u00f3, NAD, CoQ10 i fins i tot la melatonina. Dit aix\u00f2, a continuaci\u00f3, parlarem una mica m\u00e9s a fons sobre aquests antioxidants. <\/p>\n

Comen\u00e7arem amb el glutati\u00f3, <\/strong>que \u00e9s considerat l’antioxidant mestre de l’organisme, a l’ambient hospitalari \u00e9s ben conegut, es produeix a partir de N-Acetilciste\u00efna, un f\u00e0rmac indicat per a intoxicaci\u00f3 per acetaminof\u00e8n (paracetamol), aquest f\u00e0rmac a altes dosis \u00e9s for\u00e7a t\u00f2xic per al fetge i s’usa la \u201cdetoxificador hep\u00e0tic\u201d per excel\u00b7l\u00e8ncia. El glutati\u00f3 protegeix contra l’estr\u00e8s oxidatiu<\/strong>, metalls pesants i dany cel\u00b7lular, en assaigs cl\u00ednics, nivells baixos s’associen a fatiga, envelliment accelerat i deteriorament immunol\u00f2gic. A m\u00e9s de la detoxificaci\u00f3 hep\u00e0tica, tamb\u00e9 est\u00e0 implicat en la neutralitzaci\u00f3 de radicals lliures i la modulaci\u00f3 de la immunitat. Diversos estudis han demostrat que la seva disminuci\u00f3 est\u00e0 relacionada amb malalties neurodegeneratives, fatiga cr\u00f2nica i envelliment prematur. En persones amb marcadors elevats d’estr\u00e8s oxidatiu, l’\u00fas de glutati\u00f3 liposomal oral o intraven\u00f3s, segons la necessitat cl\u00ednica, ha mostrat millorar l’estat general, reduir par\u00e0metres inflamatoris i restaurar nivells d’energia. <\/p>\n

D’altra banda, el coenzim NAD\u207a<\/strong> (nicotinamida adenina dinucle\u00f2tid) \u00e9s essencial per a la reparaci\u00f3 de l’ADN, la producci\u00f3 d’energia cel\u00b7lular i l’activitat de les sirtu\u00efnes, un grup d’enzims directament vinculats amb la longevitat <\/strong>i l’estabilitat del genoma<\/strong>. S’ha comprovat que els seus nivells disminueixen de forma sostinguda amb l’edat a partir dels 40 anys, per\u00f2 poden ser restaurats amb l’administraci\u00f3 de precursors com NR (nicotinamida rib\u00f2sid) o NMN (mononucle\u00f2tid de nicotinamida), dejuni intermitent o l’exercici d’alta intensitat, per\u00f2 no tots els \u00e9ssers humans tenen la capacitat de convertir aquests precursos a NAD<\/strong>, \u00e9s per aix\u00f2 que moltes vegades es prefereix l’administraci\u00f3 intravenosa. El seu \u00fas en models animals ha perllongat la vida \u00fatil de l’animal, i en humans s’associa amb millores a l’energia, sensibilitat a la insulina i la funci\u00f3 mitocondrial, aix\u00f2 es tradueix en prevenci\u00f3 de malalties<\/strong> cr\u00f2niques i fins i tot c\u00e0ncer. <\/p>\n

Tant el NAD <\/strong>com el glutati\u00f3<\/strong> tenen un fi com\u00fa, combatre la inflamaci\u00f3, aquesta seria una altra de les claus per envellir millor. La famosa inflamaci\u00f3 de baix grau o \u201cel foc silenci\u00f3s\u201d \u00e9s un dels grans enemics de la salut. Aquest estat inflamatori no presenta problemes d’inici, per\u00f2 amb la p\u00e8rdua de certes funcions i el declivi natural de l’envelliment<\/strong> \u00e9s molt probable que aquest estat persistent i silenci\u00f3s d’activaci\u00f3 del sistema immunol\u00f2gic amb el temps s’associ\u00ef a un risc m\u00e9s alt de malalties cr\u00f2niques. Aix\u00f2 explica que ens sembli normal associar malalties a la gent gran, per\u00f2 ja \u00e9s possible revertir-ho i evitar problemes associats a l’edat, aquest fenomen ha estat anomenat \u201cinflammaging\u201d (inflammation + aging) pel seu paper en el deteriorament progressiu del sistema immunitari i metab\u00f2lic amb l’edat.<\/strong> A difer\u00e8ncia de la inflamaci\u00f3 aguda (com quan hi ha febre o dolor), aquesta forma d’inflamaci\u00f3 no es percep f\u00e0cilment, per\u00f2 es pot mesurar amb certs biomarcadors espec\u00edfics, com ara la PCR ultrasensible, la ferritina, homociste\u00efna, l’equilibri d’\u00e0cids grassos, metalls pesants, entre d’altres. <\/p>\n

A m\u00e9s, cal destacar que aquest fenomen ”inflammaging” est\u00e0 molt relacionat amb alteracions de la flora bacteriana pr\u00f2pia de l’organisme tamb\u00e9 coneguda com a microbiota<\/strong>. La p\u00e8rdua de la diversitat bacteriana, combinada amb una dieta occidental rica en ultraprocessats, pot perpetuar aquesta inflamaci\u00f3 de baix grau. Restaurar aquesta simbiosi amb prebi\u00f2tics, polifenols i, en casos espec\u00edfics, probi\u00f2tics cl\u00ednicament validats, permet no nom\u00e9s millorar la digesti\u00f3 o l’estat d’\u00e0nim, sin\u00f3 tamb\u00e9 enfortir la integritat de barreres immunol\u00f2giques i reduir biomarcadors com ara PCR ultrasensible, ferritina o interleucines proinflamat\u00f2ries. <\/p>\n

La clau seg\u00fcent que fa la difer\u00e8ncia entre un envelliment favorable i un envelliment <\/strong>desfavorable serien els marcadors metab\u00f2lics, aquests s\u00f3n clau a regular el metabolisme basal, recordem que l’envelliment s’associa a un menor metabolisme i augment de pes. Sens dubte, la protagonista seria l’hormona del creixement, l’hormona del creixement es mesura en anal\u00edtiques a trav\u00e9s del biomarcador IGF-1 (factor de creixement similar a insulina tipus 1) afavoreix la reparaci\u00f3 cel\u00b7lular, la densitat mineral \u00f2ssia i la vitalitat. Amb el pas del temps aquesta hormona<\/strong> es va perdent naturalment i comporta impacte en la salut <\/strong>general. Els nivells adequats depenen dels h\u00e0bits, per\u00f2 tamb\u00e9 del declivi d’altres hormones com s\u00f3n les hormones sexuals. Una altra hormona important quan parlem de metabolisme \u00e9s l’hormona tiro\u00efdal, especialment l’hormona T3,<\/strong> aquesta hormona regula el metabolisme basal, la massa muscular i la temperatura corporal. La funci\u00f3 tiro\u00efdal sens dubte juga un paper crucial. Per exemple, tot i que els valors de TSH puguin trobar-se dins del rang normal, una baixa conversi\u00f3 de T4 a T3 lliure pot produir s\u00edmptomes subtils d’hipofunci\u00f3: fatiga, bradips\u00edquia (boirina mental o pensament lent), sensibilitat al fred o lentitut metab\u00f2lica.En aquests casos, m\u00e9s enll\u00e0 del enfocament convencional, resulta \u00fatil considerar el context cl\u00ednic i funcional, complementant amb micronutrients com podria ser zinc i seleni o incl\u00fas realitzant una modulaci\u00f3 hormonal personalitzada si procedeix. <\/p>\n

\u00c9s molt important monitoritzar i optimitzar aquests par\u00e0metres ja que marquen la difer\u00e8ncia entre un envelliment actiu i un de prematurament limitat, tamb\u00e9 \u00e9s important destacar que quan parlem d’optimitzaci\u00f3 no significa normalitat, \u00e9s freq\u00fcent que es comentin nivells normals per a l’edat, per\u00f2 els nivells normals per a l’edat no signifiquen una funci\u00f3 \u00f2ptima i aix\u00f2 \u00e9s crucial en els nostres seguiments.<\/p>\n

En resum, la medicina de la longevitat<\/strong> no es basa en una \u00fanica intervenci\u00f3, sin\u00f3 en la integraci\u00f3 de m\u00faltiples estrat\u00e8gies que combinen nutrici\u00f3 personalitzada, optimitzaci\u00f3 hormonal, maneig de l’estr\u00e8s oxidatiu i inflamaci\u00f3, activitat f\u00edsica adaptada, microbiota i una intervenci\u00f3 molecular espec\u00edfica.<\/p>\n

El futur de l’envelliment saludable<\/strong> ja no \u00e9s una promesa; \u00e9s una possibilitat real. I comen\u00e7a coneixent la seva salut des de dins amb el mesurament dels seus marcadors en un centre especialitzat. A les nostres cl\u00edniques us oferim una valoraci\u00f3 integral, amb enfocament en longevitat, metabolisme, microbiota i salut hormonal. <\/p>\n

\n

BIBLIOGRAFIA<\/p>\n

(1) Martens et al. (2018) \u2013 Chronic nicotinamide riboside supplementation is well-tolerated and elevates NAD+<\/sup>in healthy middle-aged and older adults. Nat Commun. 2018 Mar 29;9(1):1286 <\/p>\n

(2) Sinha et al. (2017) \u2013 <\/strong>Oral supplementation with liposomal glutathione elevates body stores of glutathione and markers of immune function. Eur J Clin Nutr. 2018 Jan;72(1):105-111 <\/p>\n

(3) Carlijn ME Remie, Kay HM Roumans, et.al. Nicotinamide riboside supplementation alters body composition and skeletal muscle acetylcarnitine concentrations in healthy obese humans, The American Journal of Clinical Nutrition, Volume 112, Issue 2, 2020, Pages 413-426, ISSN 0002-9165. <\/p>\n

(4) Cisneros B, et al. Immune system modulation in aging: Molecular mechanisms and therapeutic targets. Front Immunol. 2022 Dec 15;13:1059173 <\/p>\n

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Es pot “modular” l’envelliment? La resposta \u00e9s: S\u00ed. I la ci\u00e8ncia ens d\u00f3na cada cop m\u00e9s eines per fer-ho de forma segura i efectiva. Durant anys es va assumir que l’envelliment era un proc\u00e9s inevitable, dictat exclusivament per la gen\u00e8tica i el pas del temps. Avui sabem que no \u00e9s aix\u00ed. La investigaci\u00f3 biom\u00e8dica ha […]<\/p>\n","protected":false},"author":30,"featured_media":29219,"comment_status":"closed","ping_status":"closed","sticky":false,"template":"","format":"standard","meta":{"_acf_changed":false,"inline_featured_image":false,"footnotes":""},"categories":[5198],"tags":[5148,5236,5143,5149,5232,5144,5233,5140,5171,5235,5231,5230,5234],"class_list":["post-29258","post","type-post","status-publish","format-standard","has-post-thumbnail","hentry","category-neolife-ca","tag-antioxidant-ca","tag-envellir","tag-estres-oxidatiu","tag-glutatio","tag-igf-1-ca","tag-inflamacio","tag-inflammaging-ca","tag-longevitat","tag-microbiota-ca","tag-molecular-ca","tag-nad-ca","tag-qualitat-de-vida","tag-salut-hormonal"],"acf":[],"yoast_head":"\nLongevitat intel\u00b7ligent<\/title>\n<meta name=\"description\" content=\"Es pot \u201cmodular\u201d l'envelliment? La resposta \u00e9s: Si. 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